A group of scientists from Mount Sinai Hospital of New York, one of the most relevant centers in cardiac and vascular research and surgery, has revealed the way in which brain and heart communicate with each other through the immune system to promote sleep and recovery after a serious cardiovascular event.
Yes, like a heart attack. The conclusions, published in the journal Naturethey stand out the importance of increasing sleep after a myocardial infarction and suggest that getting enough sleep should be one of the vital points of the clinical treatment and care after an event of such dimensions.
Even in the intensive care units (ICU), where the dream is interrupted frequentlyalong with cardiac rehabilitation. Although it has generated some controversy on social networks, the investigation has left no room for possible interpretations.
In depth
Specifically, the researchers first used mouse models to discover this phenomenon: induced heart attacks in half of the animals and carried out high-resolution cellular and imaging analyzes.
Furthermore, they used wireless electroencephalography devices implantables to record electrical signals from their brains and analyze sleep patterns. After the heart attack, They perfectly appreciated that it tripled the slow wave dream.
When experts studied the brains of stroked mice, they discovered that immune cells known as “monocytes“were recruited from the blood to the brain and used the TNF protein to activate neurons in an area of the brain called the thalamus, which caused increased sleep.
As detailed by the investigators, this occurred in the hours following the cardiac event and none of this happened in the mice that did not suffer heart attacks.
More details
They then used sophisticated methods to manipulate neural signaling in the brain. TNF in the thalamus and discovered that the sleeping brain uses the nervous system to send signals to the heart to reduce cardiac stress, promote healing and decrease cardiac inflammation after a heart attack and therefore improve recovery.
To better identify the role of increased sleep after a heart attack, the researchers also interrupted the sleep of some of the mice. In this way, they verified that mice with interrupted sleep after a heart attack presented a increased heart sympathetic stress responses and inflammation.
This caused a recovery and healing slower compared to mice with uninterrupted sleep. The research team at Mount Sinai Hospital also ran several studies in humans.
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